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文  献 9)Matsuzawa-Ishimoto Y, Shono Y, Gomez LE, Hubbard-Lucey VM, Cammer M, Neil J, et al. A u t o p h a g y p r o t e i n A T G 1 6 L 1 p r e v e n t s necroptosis in the intestinal epithelium. J Exp Med 2017; 214(12): 3687-705.10)Matsuzawa-Ishimoto Y, Hine A, Shono Y, Rudensky E, Lazrak A, Yeung F, et al. An intestinal organoid-based platform that recreates susceptibility to T-cell-mediated tissue injury. Blood 2020; 135(26): 2388-401.11)Matsuzawa-Ishimoto Y, Yao X, Koide A, Ueberheide BM, Axelrad JE, Reis BS, et al. The γδ I E L e f f e c t o r A P I 5 m a s k s g e n e t i c susceptibility to Paneth cell death. Nature 2022; 610(7932): 547-54.12)Harvey RF, Bradshaw JM. A simple index of Crohn's-disease activity. Lancet 1980; 1(8167): 514.13)Takenaka K, Ohtsuka K, Kitazume Y, Nagahori M, Fujii T, Saito E, et al. Comparison of magnetic resonance and balloon enteroscopic examination of the small intestine in patients with Crohn's disease. Gastroenterology 2014; 147(2): 334-42. e3. 14)Kitazume Y, Fujioka T, Takenaka K, Oyama J, Ohtsuka K, Fujii T, et al. Crohn disease: A 5-point MR enterocolonography classification using enteroscopic findings. AJR Am J Roentgenol 2019; 212(1): 67-76.15)Fujii S, Suzuki K, Kawamoto A, Ishibashi F, Nakata T, Murano T, et al. PGE2 is a direct and robust mediator of anion/fluid secretion by human intestinal epithelial cells. Sci Rep 2016; 6: 36795.16)Fujii M, Matano M, Toshimitsu K, Takano A, Mikami Y, Nishikori S, et al. Human intestinal organoids maintain self-renewal capacity and cellular diversity in niche-inspired culture condition. Cell Stem Cell 2018; 23(6): 787-93. e6.17)Wang Z, Shen J. The role of goblet cells in Crohn's disease. Cell Biosci 2024; 14(1): 43.18)Date S, Sato T. Mini-gut organoids: reconstitution of the stem cell niche. Annu Rev Cell Dev Biol 2015; 31: 269-89. 1)Pathiyil MM, Jena A, Venkataramana Raju AK, Omprakash TA, Sharma V, Sebastian S. et al. Representation and reporting of diverse groups i n r a n d o m i s e d c o n t r o l l e d t r i a l s o f pharmacological agents in inflammatory bowel disease: a systematic review. Lancet Gastroenterol Hepatol 2023; 8(12): 1143-51. 2)Plichta DR, Graham DB, Subramanian S, Xavier RJ. Therapeutic Opportunities in Inflammatory Bowel Disease: Mechanistic Dissection of Host-Microbiome Relationships. Cell 2019; 178(5): 1041-56. 3)Goyette P, Labbé C, Trinh TT, Xavier RJ, Rioux JD. et al. Molecular pathogenesis of inflammatory bowel disease: genotypes, phenotypes and personalized medicine. Ann Med 2007; 39(3): 177-99. 4)Cadwell K, Liu JY, Brown SL, Miyoshi H, Loh J, Lennerz JK, et al. A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells. Nature 2008; 456(7219): 259-63. 5)Hampe J, Franke A, Rosenstiel P, Till A, Teuber M, Huse K, et al. A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1. Nat Genet 2007; 39(2): 207-11. 6)Cadwell K, Patel KK, Maloney NS, Liu TC, Ng AC, Storer CE, et al. Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine. Cell 2010; 141(7): 1135-45. 7)Clevers HC, Bevins CL. Paneth cells: maestros of the small intestinal crypts. Annu.Rev. Physiol 2013; 75; 289–311. 8)VanDussen KL, Liu TC, Li D, Towfic F, Modiano N, Winter R, et al. Genetic variants synthesize to produce paneth cell phenotypes that define subtypes of Crohn's disease. Gastroenterology 2014; 146(1): 200-9.日本人のクローン病患者におけるパネート細胞異常の解析153

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