文 献 4） Yang X, Liu J, Liu W, Wu H, Wei Y, Guo X, et al. circFAM193B interaction with PRMT6 r e g u l a t e s A M L l e u k e m i a s t e m c e l l s chemoresistance through altering the oxidative metabolism and lipid peroxidation. Leukemia 2024; 38: 1057-71. 5） Ikeda J, Kunimoto H, Saito Y, Tsujimoto S, T a k e u c h i M , M i u r a A , e t a l . M e t a b o l i c reprogramming by PRDM16 drives cytarabine r e s i s t a n c e i n a c u t e m y e l o i d l e u k e m i a . Haematologica 2025; 110: 2647-60. 1 ）Shiba N, Ohki K, Kobayashi T, Hara Y, Yamato G, Tanoshima R, et al. High PRDM16 expression identifies a prognostic subgroup of pediatric acute myeloid leukaemia correlated to FLT3-ITD, KMT2A-PTD, and NUP98-NSD1: t h e r e s u l t s o f t h e J a p a n e s e P a e d i a t r i c Leukaemia/Lymphoma Study Group AML-05 trial. Br J Haematol 2016; 172: 581-91. 2） Amaya ML, Inguva A, Pei S, Jones C, Krug A, Ye H, et al. The STAT3-MYC axis promotes survival of leukemia stem cells by regulating SLC1A5 and oxidative phosphorylation. Blood 2022; 139: 584-96. 3） Aroua N, Boet E, Ghisi M, Nicolau-Travers ML, Saland E, Gwilliam R, et al. Extracellular ATP a n d C D 3 9 a c t i v a t e c A M P - m e d i a t e d mitochondrial stress response to promote cytarabine resistance in acute myeloid leukemia. Cancer Discov 2020; 10: 1544-65.PRDM16による代謝リプログラミングは白血病細胞のシタラビン耐性を誘導する131
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